用户:S22668866/save

存档旧内容

==

此用户的封禁已被其他管理员复检，申诉被拒绝。

此用户的：封禁记录 · 用户贡献(已删除) · 日志(帐户创建者 · 中央认证 · 滥用过滤器) · 更改封禁设定 · 授予IPBE · 解除封禁

请求复检的理由或相关提问：

我并没有提供不实资料这个封锁是不合理的

注意：

滥用此模板或申诉渠道的用户，可能被延长封禁或被禁止编辑其用户讨论页。
如您希望与管理员就本次封禁作出讨论，请于本模板外展开。

此封禁决定的复检已有以下结论：

基于下列理由，本人决定拒绝这位用户的申诉。其他管理员可再复检此用户的封禁，但在没有更好的理由前，请勿轻易推翻之前的决定。
重复
处理人： Stang^★★ 2019年11月30日 (六) 13:26 (UTC)

==

此使用者目前已被封锁。以下为最近的封锁纪录以供参考：

   2019年11月28日 (四) 18:21 Mys 721tx 討論 貢獻已封鎖S22668866 討論 貢獻期限為1 週（停用帳號建立） （屢次增加不實資料）

我认为这是一个严重的毁谤

关于毒品和快乐中枢的相关资料非常多繁衍会刺激快乐中枢的资料也同时有说到

http://www.epochtimes.com/b5/7/2/1/n1610828.htm

https://blog.xuit.net/upvna/twblog/193966822-%E8%85%A6%E9%83%A8%E7%9A%84%E5%BF%AB%E6%A8%82%E4%B8%AD%E6%A8%9E

http://enc.nutn.edu.tw/Page/Index/%E6%AF%92%E5%93%81%E5%88%86%E7%B4%9A%E4%BB%8B%E7%B4%B9

https://antidrug.moj.gov.tw/lp-1211-2.html 甚至政府的官方网站也有

以及医学院公开发表的医学报告也有 https://ksph.kcg.gov.tw/walking04.htm

https://www.therecoveryvillage.com/heroin-addiction/heroin-affects-nervous-system/#gref 海洛因影响大脑的关键区域与愉悦途径有关，包括额叶皮层和伏隔核。

我觉得这种不实指控非常扯

没有经过考究就指控我提供不实资料毒品相关的论文各种考据各项论文都有

我在这里申诉这是一项严重的毁谤毁谤我提供不实资料

况且不要说哪里的论文连不同语言的维基百科也有提到 https://en.m.wikipedia.org/wiki/Reward_system

关于快乐中枢和毒品和繁衍的关联性我告诉你你们怎不把每个条目都删光光

这种行为等同于正在封锁一个消息没有经过考究就连不同语言的WIKI都有提到快乐中枢和毒品和繁衍的关联性

我强烈谴责这种封锁消息的行为且我要求恢复我所发布的资料

相关依据上面都有就连不同语言的维基百科里都有写到 https://en.m.wikipedia.org/wiki/Reward_system 在WIKI中相关来源也都有没有任何理由可以说我提供不实资料我非常强烈谴责这个指控

此用户的封禁已被其他管理员复检，申诉被拒绝。

此用户的：封禁记录 · 用户贡献(已删除) · 日志(帐户创建者 · 中央认证 · 滥用过滤器) · 更改封禁设定 · 授予IPBE · 解除封禁

请求复检的理由或相关提问：

我并没有提供不实资料这个封锁是不合理的

注意：

滥用此模板或申诉渠道的用户，可能被延长封禁或被禁止编辑其用户讨论页。
如您希望与管理员就本次封禁作出讨论，请于本模板外展开。

此封禁决定的复检已有以下结论：

基于下列理由，本人决定拒绝这位用户的申诉。其他管理员可再复检此用户的封禁，但在没有更好的理由前，请勿轻易推翻之前的决定。
您在被封禁之前并没有添加任何可以支持您添加内容的来源至条目中。如果希望添加，请自行在封禁期结束之后进行添加。封禁不予解除。
处理人： Stang^★★ 2019年11月30日 (六) 13:26 (UTC)

没有经过考究　　就说我提供不实资料　　这就是毁谤　　

我现在证据我都附上了　　这不实的指控　居然没有解除

要求来源可以　　但是不实的指控我提供不实资料

这就是毁谤　　我强烈谴责这种行为

管理员没有进行考究　　就对使用者毁谤

这是非常恶劣的指控　已经侮辱了维基百科的使用者

我也拿出证据上诉了　　居然还是指控我提供不实资料　　这是不合理的

再者本来就要进行考究　　才能扣别人帽子　现在就是对于我的毁谤

我非常强烈谴责这项行为

此用户的封禁已被其他管理员复检，申诉被拒绝。

此用户的：封禁记录 · 用户贡献(已删除) · 日志(帐户创建者 · 中央认证 · 滥用过滤器) · 更改封禁设定 · 授予IPBE · 解除封禁

请求复检的理由或相关提问：

没有经过考究　　就说我提供不实资料　　这就是毁谤　　

我现在证据我都附上了　　这不实的指控　居然没有解除要求来源可以　　但是不实的指控我提供不实资料这就是毁谤　　我强烈谴责这种行为管理员没有进行考究　　就对使用者毁谤这是非常恶劣的指控　已经侮辱了维基百科的使用者我也拿出证据上诉了　　居然还是指控我提供不实资料　　这是不合理的再者本来就要进行考究　　才能扣别人帽子　现在就是对于我的毁谤

我非常强烈谴责这项行为

注意：

滥用此模板或申诉渠道的用户，可能被延长封禁或被禁止编辑其用户讨论页。
如您希望与管理员就本次封禁作出讨论，请于本模板外展开。

此封禁决定的复检已有以下结论：

基于下列理由，本人决定拒绝这位用户的申诉。其他管理员可再复检此用户的封禁，但在没有更好的理由前，请勿轻易推翻之前的决定。
经过复查，此前的封禁决定与对封禁申诉的拒绝没有不合理之处，您也未能提供新的论述或作出真诚悔改。基于以上考量，决定拒绝您的封禁申诉。如果您继续滥用封禁申诉途径，您可能被一并剥夺讨论页的编辑权力。因此，如果您决定再进行一次封禁申诉，希望您能够做好思想上的准备并真诚悔过，而不是重复一样的观点。
处理人：クオン·🀢🀣🀤🀥·翡翠·鵺鳥·十姉妹·夜啼鳥 2019年11月30日 (六) 19:24 (UTC)

上面是被封锁一周

以下是被封锁一个月的申诉

请还我一个公道谢谢

此用户的封禁已被其他管理员复检，申诉被接受。

此用户的：封禁记录 · 用户贡献(已删除) · 日志(帐户创建者 · 中央认证 · 滥用过滤器) · 更改封禁设定 · 授予IPBE · 解除封禁

请求复检的理由或相关提问：

我已经更改了与上次不同的来源

且我已经在摘要里面附注来源中有提到 "海洛因影响大脑的关键区域与奖励或愉悦途径有关，包括额叶皮层和伏隔核。这些回路对于自然的奖励非常重要，例如在您用餐或做爱时会得到什么。" 来源是用以下这个

https://www.therecoveryvillage.com/heroin-addiction/heroin-affects-nervous-system/#gref

请管理员还我一个公道谢谢

我本身是化学系的，毒品也是常出现在化学文献里，像是毒品化学式，毒品化学鉴识等，毒品相关的国内外论文我看过不少，

我只是想增加毒品条目里相当重要的一个重点，

但是屡次被这个管理员封锁User:Mys_721tx，我也觉得很疲累了，希望可以还我一个公道，而且我附注的这来源就完全有提到"海洛因影响大脑的关键区域与奖励或愉悦途径有关，包括额叶皮层和伏隔核。这些

回路对于自然的奖励非常重要，例如在您用餐或做爱时会得到什么。"

我也附上来源，而且加强备注了，结果还是遭到封锁

我希望可以跟管理员对话，如果觉得我增加的是不实资料，也要经过讨论反驳并且附上来源证实，而不是直接封锁我，

我都已经附上来源，而且确切的有提到"海洛因(鸦片相关类药物)影响大脑的关键区域与奖励或愉悦途径有关。"

该封禁的查封ID是#相关的IP封锁区段：。--WW（留言） 2019年12月9日 (一) 14:51 (UTC)

注意：

滥用此模板或申诉渠道的用户，可能被延长封禁或被禁止编辑其用户讨论页。
如您希望与管理员就本次封禁作出讨论，请于本模板外展开。

此封禁决定的复检已有以下结论：

基于下列理由，本人决定接受这位用户的申诉并调整封禁。其他管理员可再复检此用户的封禁，但在没有更好的理由前，请勿轻易推翻之前的决定。
我接受这个封禁申诉，因用户是新手，可能不易理解维基的方针。此外，关于绕过封禁，我接纳用户指出，“这些并不是我的编辑，而且1.65.160.18是香港IP”的解释；另，于该用户作出该声明后，该IP仍有编辑，故不大可能是用户在知悉因绕过封禁而被延长封禁，再以IP作出相关编辑。是故，接纳其此部分申诉。因此提前解除封禁。

希望您日后可理解维基百科相关的方针。如您需要协助，维基百科:小天使工作小组、维基百科:新手会的人相信乐意协助您，我深信他们会协助您理解维基百科。-千村狐兔（留言） 2020年1月3日 (五) 15:01 (UTC)
处理人：请签署

(：)回应：请阁下详细说明该来源中哪段内容有提到你所增加的内容。谢谢。--SCP-2000 2019年12月10日 (二) 20:16 (UTC)
- (！)意见申诉者只是第二次因违反方针而被封禁，且申诉者只是在单一条目内进行违反方针之行为，再者其他人及该名管理员未有向他发出警告，未能让他完全得知其情况，故私认为没有封一个月的必要及应予缩减封禁时间。--SCP-2000 2019年12月10日 (二) 20:16 (UTC)
  "这是一项没有理由的删除相关毒品的论文国内外都有我写的这段就是毒品的真相"、 "已阅读内容"。具备检查页面历史的能力应当是讨论封禁必要性资格的必要条件。-Mys_721tx（留言） 2019年12月10日 (二) 20:38 (UTC)
  - 谢谢阁下意见，基本上每次讨论我会先看当事人的贡献及页面历史。另外，私不认为未经警告下，就直接进行封禁是合理及符合封禁方针的。谢谢。--SCP-2000 2019年12月10日 (二) 21:34 (UTC)
    - 翻了一下条目历史，有个层级2级的警告。Itcfangye（留言） 2019年12月11日 (三) 05:36 (UTC)

我使用的来源

https://www.therecoveryvillage.com/heroin-addiction/heroin-affects-nervous-system/#gref

来源中这段有说到我所增加的内容

The key areas of the brain that are impacted by heroin are related to the reward or pleasure pathways and they include the frontal cortex and the nucleus accumbens. These circuits are important for natural rewards such as what comes when you enjoy a meal or have sex.--WW（留言）

我想补充一点，其实在毒品的不同语言维基百科中，都有十分可靠的来源，显现了我所增加的内容，举例来说，

https://en.m.wikipedia.org/wiki/Reward_system ，这里面有提到，

The brain structures that compose the reward system are located primarily within the cortico-basal ganglia-thalamo-cortical loop，

The reward system is a group of neural structures responsible for incentive salience (ie, motivation and "wanting"

particularly ones which involve pleasure as a core component

有提到大脑中枢包含着奖励系统，负责刺激欲望，并且以愉悦感为核心

Primary rewards are a class of rewarding stimuli which facilitate the survival of one's self and offspring , and include homeostatic (eg, palatable food ) and reproductive (eg, sexual contact and parental investment ) rewards.^[1]

主要奖励是一类奖励性刺激，可促进一个人的自身和后代的生存，并包括体内平衡（例如，美味食品）和生殖（例如，性行为和亲代投资）奖励。

The hedonic hotspots located in the anterior OFC and posterior insula have been demonstrated to respond to orexin and opioids, as has the overlapping hedonic coldspot in the anterior insula and posterior OFC.[29] On the other hand, the parabrachial nucleus hotspot has only been demonstrated to respond to benzodiazepine receptor agonists.

已证明位于OFC前部和后岛的享乐性热点对食欲素和鸦片类药物有反应，在前岛区和后部OFC中有重叠的享乐性热点。^[2]

上述都有可靠的来源且，在条目所增加的内容，我是依据其他语言的毒品维基百科内容过来的，

如果认为我增加不实资料，我认为应该要提出可靠的依据来，且对其他语言WIKI也做出反应才对

然而也要有足够的依据反驳上述来源--WW（留言）

参考资料

^ Schultz W (2015). "Neuronal reward and decision signals: from theories to data". Physiological Reviews. 95 (3): 853–951. doi:10.1152/physrev.00023.2014. PMC 4491543. PMID 26109341. "Rewards in operant conditioning are positive reinforcers. ... Operant behavior gives a good definition for rewards. Anything that makes an individual come back for more is a positive reinforcer and therefore a reward. Although it provides a good definition, positive reinforcement is only one of several reward functions. ... Rewards are attractive. They are motivating and make us exert an effort. ... Rewards induce approach behavior, also called appetitive or preparatory behavior, sexual behavior, and consummatory behavior. ... Thus any stimulus, object, event, activity, or situation that has the potential to make us approach and consume it is by definition a reward. ... Rewarding stimuli, objects, events, situations, and activities consist of several major components. First, rewards have basic sensory components (visual, auditory, somatosensory, gustatory, and olfactory) ... Second, rewards are salient and thus elicit attention, which are manifested as orienting responses (FIGURE 1, middle). The salience of rewards derives from three principal factors, namely, their physical intensity and impact (physical salience), their novelty and surprise (novelty/surprise salience), and their general motivational impact shared with punishers (motivational salience). A separate form not included in this scheme, incentive salience, primarily addresses dopamine function in addiction and refers only to approach behavior (as opposed to learning) ... Third, rewards have a value component that determines the positively motivating effects of rewards and is not contained in, nor explained by, the sensory and attentional components (FIGURE 1, right). This component reflects behavioral preferences and thus is subjective and only partially determined by physical parameters. Only this component constitutes what we understand as a reward. It mediates the specific behavioral reinforcing, approach generating, and emotional effects of rewards that are crucial for the organism’s survival and reproduction, whereas all other components are only supportive of these functions. ... Rewards can also be intrinsic to behavior (31, 546, 547). They contrast with extrinsic rewards that provide motivation for behavior and constitute the essence of operant behavior in laboratory tests. Intrinsic rewards are activities that are pleasurable on their own and are undertaken for their own sake, without being the means for getting extrinsic rewards. ... Intrinsic rewards are genuine rewards in their own right, as they induce learning, approach, and pleasure, like perfectioning, playing, and enjoying the piano. Although they can serve to condition higher order rewards, they are not conditioned, higher order rewards, as attaining their reward properties does not require pairing with an unconditioned reward. ... These emotions are also called liking (for pleasure) and wanting (for desire) in addiction research (471) and strongly support the learning and approach generating functions of reward."
^ Yager LM, Garcia AF, Wunsch AM, Ferguson SM (August 2015). "The ins and outs of the striatum: Role in drug addiction". Neuroscience. 301: 529–541. doi:10.1016/j.neuroscience.2015.06.033. PMC 4523218. PMID 26116518. "[The striatum] receives dopaminergic inputs from the ventral tegmental area (VTA) and the substantia nigra (SNr) and glutamatergic inputs from several areas, including the cortex, hippocampus, amygdala, and thalamus (Swanson, 1982; Phillipson and Griffiths, 1985; Finch, 1996; Groenewegen et al., 1999; Britt et al., 2012). These glutamatergic inputs make contact on the heads of dendritic spines of the striatal GABAergic medium spiny projection neurons (MSNs) whereas dopaminergic inputs synapse onto the spine neck, allowing for an important and complex interaction between these two inputs in modulation of MSN activity ... It should also be noted that there is a small population of neurons in the [nucleus accumbens] NAc that coexpress both D1 and D2 receptors, though this is largely restricted to the NAc shell (Bertran- Gonzalez et al., 2008). ... Neurons in the NAc core and NAc shell subdivisions also differ functionally. The NAc core is involved in the processing of conditioned stimuli whereas the NAc shell is more important in the processing of unconditioned stimuli; Classically, these two striatal MSN populations are thought to have opposing effects on basal ganglia output. Activation of the dMSNs causes a net excitation of the thalamus resulting in a positive cortical feedback loop; thereby acting as a 'go’ signal to initiate behavior. Activation of the iMSNs, however, causes a net inhibition of thalamic activity resulting in a negative cortical feedback loop and therefore serves as a 'brake’ to inhibit behavior ... there is also mounting evidence that iMSNs play a role in motivation and addiction (Lobo and Nestler, 2011; Grueter et al., 2013). For example, optogenetic activation of NAc core and shell iMSNs suppressed the development of a cocaine CPP whereas selective ablation of NAc core and shell iMSNs ... enhanced the development and the persistence of an amphetamine CPP (Durieux et al., 2009; Lobo et al., 2010). These findings suggest that iMSNs can bidirectionally modulate drug reward. ... Together these data suggest that iMSNs normally act to restrain drug-taking behavior and recruitment of these neurons may in fact be protective against the development of compulsive drug use."

希望您在编辑时，注意增添内容需要有可靠文献支持您的论点。-千村狐兔（留言） 2019年12月23日 (一) 13:53 (UTC)

有，我已经贴了可靠文献了，请问是否有看到上面贴的那两个文献---->[[1]][[2]] WW（留言）

您是否有绕过封禁的行为?-千村狐兔（留言） 2019年12月29日 (日) 11:46 (UTC)

@Mys 721tx：请问S22668866君的傀儡在哪个条目进行编辑？谢谢。--SCP-2000 _{按此申诉本人判断} 2019年12月29日 (日) 11:59 (UTC)

Special:Contributions/1.65.160.18。-Mys_721tx（留言） 2019年12月30日 (一) 02:13 (UTC)

看起来是

不相关，S22668866君是一次过增加大量原创研究内容，且以段落之方式撰写。然而ip君是一次过增加较少原创研究内容，及以点列方式撰写。再者S22668866君未曾编辑过有关香港及历史的条目。--SCP-2000 _{按此申诉本人判断} 2019年12月30日 (一) 02:30 (UTC)

ul/dl/ol在wikitext上仅一字符之差。分段及行文方式亦与本页面文字相似。-Mys_721tx（留言） 2019年12月30日 (一) 05:08 (UTC)

@Mys 721tx： S22668866君在这里的行文方式为一行中有几句句子，而ip君的行文方式为一行中有几个词语或短句。再者正常编辑时，大多会使用「:#*」这些符号，而非使用ul/dl/ol。--SCP-2000 _{按此申诉本人判断} 2019年12月30日 (一) 05:22 (UTC)

Special:Contributions/1.65.160.18这些并不是我的编辑，而且1.65.160.18是香港IP，跟我完全不同WW（留言）

cc@Manchiu： --SCP-2000 _{按此申诉本人判断} 2020年1月1日 (三) 09:01 (UTC)

知悉其解释。应予新手善意推定。-千村狐兔（留言） 2020年1月3日 (五) 14:54 (UTC)

[1] Schultz W (2015). "Neuronal reward and decision signals: from theories to data". Physiological Reviews. 95 (3): 853–951. doi:10.1152/physrev.00023.2014. PMC 4491543. PMID 26109341. "Rewards in operant conditioning are positive reinforcers. ... Operant behavior gives a good definition for rewards. Anything that makes an individual come back for more is a positive reinforcer and therefore a reward. Although it provides a good definition, positive reinforcement is only one of several reward functions. ... Rewards are attractive. They are motivating and make us exert an effort. ... Rewards induce approach behavior, also called appetitive or preparatory behavior, sexual behavior, and consummatory behavior. ... Thus any stimulus, object, event, activity, or situation that has the potential to make us approach and consume it is by definition a reward. ... Rewarding stimuli, objects, events, situations, and activities consist of several major components. First, rewards have basic sensory components (visual, auditory, somatosensory, gustatory, and olfactory) ... Second, rewards are salient and thus elicit attention, which are manifested as orienting responses (FIGURE 1, middle). The salience of rewards derives from three principal factors, namely, their physical intensity and impact (physical salience), their novelty and surprise (novelty/surprise salience), and their general motivational impact shared with punishers (motivational salience). A separate form not included in this scheme, incentive salience, primarily addresses dopamine function in addiction and refers only to approach behavior (as opposed to learning) ... Third, rewards have a value component that determines the positively motivating effects of rewards and is not contained in, nor explained by, the sensory and attentional components (FIGURE 1, right). This component reflects behavioral preferences and thus is subjective and only partially determined by physical parameters. Only this component constitutes what we understand as a reward. It mediates the specific behavioral reinforcing, approach generating, and emotional effects of rewards that are crucial for the organism’s survival and reproduction, whereas all other components are only supportive of these functions. ... Rewards can also be intrinsic to behavior (31, 546, 547). They contrast with extrinsic rewards that provide motivation for behavior and constitute the essence of operant behavior in laboratory tests. Intrinsic rewards are activities that are pleasurable on their own and are undertaken for their own sake, without being the means for getting extrinsic rewards. ... Intrinsic rewards are genuine rewards in their own right, as they induce learning, approach, and pleasure, like perfectioning, playing, and enjoying the piano. Although they can serve to condition higher order rewards, they are not conditioned, higher order rewards, as attaining their reward properties does not require pairing with an unconditioned reward. ... These emotions are also called liking (for pleasure) and wanting (for desire) in addiction research (471) and strongly support the learning and approach generating functions of reward."

[2] Yager LM, Garcia AF, Wunsch AM, Ferguson SM (August 2015). "The ins and outs of the striatum: Role in drug addiction". Neuroscience. 301: 529–541. doi:10.1016/j.neuroscience.2015.06.033. PMC 4523218. PMID 26116518. "[The striatum] receives dopaminergic inputs from the ventral tegmental area (VTA) and the substantia nigra (SNr) and glutamatergic inputs from several areas, including the cortex, hippocampus, amygdala, and thalamus (Swanson, 1982; Phillipson and Griffiths, 1985; Finch, 1996; Groenewegen et al., 1999; Britt et al., 2012). These glutamatergic inputs make contact on the heads of dendritic spines of the striatal GABAergic medium spiny projection neurons (MSNs) whereas dopaminergic inputs synapse onto the spine neck, allowing for an important and complex interaction between these two inputs in modulation of MSN activity ... It should also be noted that there is a small population of neurons in the [nucleus accumbens] NAc that coexpress both D1 and D2 receptors, though this is largely restricted to the NAc shell (Bertran- Gonzalez et al., 2008). ... Neurons in the NAc core and NAc shell subdivisions also differ functionally. The NAc core is involved in the processing of conditioned stimuli whereas the NAc shell is more important in the processing of unconditioned stimuli; Classically, these two striatal MSN populations are thought to have opposing effects on basal ganglia output. Activation of the dMSNs causes a net excitation of the thalamus resulting in a positive cortical feedback loop; thereby acting as a 'go’ signal to initiate behavior. Activation of the iMSNs, however, causes a net inhibition of thalamic activity resulting in a negative cortical feedback loop and therefore serves as a 'brake’ to inhibit behavior ... there is also mounting evidence that iMSNs play a role in motivation and addiction (Lobo and Nestler, 2011; Grueter et al., 2013). For example, optogenetic activation of NAc core and shell iMSNs suppressed the development of a cocaine CPP whereas selective ablation of NAc core and shell iMSNs ... enhanced the development and the persistence of an amphetamine CPP (Durieux et al., 2009; Lobo et al., 2010). These findings suggest that iMSNs can bidirectionally modulate drug reward. ... Together these data suggest that iMSNs normally act to restrain drug-taking behavior and recruitment of these neurons may in fact be protective against the development of compulsive drug use."

[1]

[2]